cross-posted from: https://lemmy.zip/post/1892119
Archived version: https://archive.ph/qgUwg
Archived version: https://web.archive.org/web/20230825150703/https://www.euronews.com/next/2023/08/23/risk-of-long-covid-persists-two-years-post-infection-study-shows
surprising absolutely no rational person at all, covid will fuck you up, so the best protection is never contracting it in the first place, with all the precautions necessary to ensure that you don’t, like masks, distancing, and paying fucking attention to the infection levels in your community. i know, bummer, right? you know what’s a bigger bummer … covid induced illness that you carry around like fucking luggage for the rest of your shortened life
Not gonna hide my face and abuse crowds my whole life
Ok… Go catch long covid then lmao
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I wonder how is this measured, because I think by now we all have been exposed to COVID, one way or another. Sometimes without knowing. What they could be looking is simply people with a weaker immune system vs stronger immune system/ less preconditions vs more preconditions. I didn’t have time to analyse the article/paper right now but with a quick read I don’t think I noticed explanation how they control against that.
I think just a sample size negates “weaker immune system vs stronger immune system/ less preconditions vs more preconditions”.
We built a cohort of 138,818 individuals with SARS-CoV-2 infection and 5,985,227 noninfected control group from the US Department of Veterans Affairs and followed them for 2 years to estimate the risks of death and 80 prespecified postacute sequelae of COVID-19 (PASC) according to care setting during the acute phase of infection.
Why would the sample size negate it?
If you assume that people with stronger immune system and less preconditions are less likely to catch COVID, or notice having caught it, surely the sample size does nothing to prevent this bias?
